By Lucy L. Chen (editor), Tsung O. Cheng (editor)
This publication bargains finished updates of present wisdom in acupuncture perform and study. The booklet is meant for physicians, acupuncturists, scientific scholars and different healthcare prone who're attracted to acupuncture remedy and research.
The chapters are divided into 4 different types: 1) Acupuncture study; 2) New advancements in Acupuncture; three) Acupuncture remedy for medical stipulations and four) evaluate and Accessibility of Acupuncture treatment.
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69] LeDoux J. The emotional brain, fear, and the amygdala. Cell Mol Neurobiol 2003;23(4-5)727-738.  Yim YK, Lee H, Hong KE, et al. Electro-acupuncture at acupoint ST36 reduces inflam‐ mation and regulates immune activity in Collagen-Induced. Evidence-based comple‐ mentary and alternative medicine : eCAM 2007;451-57.  Yu Y, Kasahara T, Sato T, et al. Role of endogenous interferon-γ on the enhancement of splenic NK cell activity by electroacupuncture stimulation in mice. Journal of neuro‐ immunology 1998;90176-186.
NO formation in acupunctured forearm: HPCL study Prior to receiving acupuncture, there were no differences in regard to plasma concentrations of nitrate plus nitrite between the real and sham acupuncture groups (Figure 4-B). After acu‐ puncture as well, no significant changes in plasma concentrations of nitrate plus nitrite were noted in either the non-treated forearm in subjects who underwent real acupuncture or 41 42 Acupuncture in Modern Medicine those that underwent sham treatment. However, that concentration in the acupunctured forearm was significantly increased as compared with both the pre-acupuncture value and values obtained for the opposite non-treated and sham-acupunctured forearms at 5 and 60 minutes after real acupuncture treatment.
Acid-sensing ion channel 3 mediates peripher‐ al anti-hyperalgesia effects of acupuncture in mice inflammatory pain. J Biomed Sci 2011;1882.  Groth R, Aanonsen L. Spinal brain-derived neurotrophic factor (BDNF) produces hy‐ peralgesia in normal mice while antisense directed against either BDNF or trkB, pre‐ vent inflammation-induced hyperalgesia. Pain 2002;100(1-2)171-181.  Mamet J, Lazdunski M, Voilley N. How nerve growth factor drives physiological and inflammatory expressions of acid-sensing ion channel 3 in sensory neurons.