By Hans Hoff, Kurt Jellinger (auth.), Igor Klatzo, Franz Seitelberger (eds.)
The Symposium on mind Edema has been geared up via the Osterreichische Arbeitsgemeinschaft fur Neuropathologie and via the matter fee for Neuropathology of the realm Federa tion of Neurology. we want to specific our because of Dr. L. van Bogaert, president of the W.F.N., for his nice curiosity within the layout of this convention and for his lively aid. one of many goals ofthe challenge fee for Neuropathology has been to give a contribution to the advances in a number of difficulties that are changing into ripe for elucidation by means of specialists in numerous fields. the assumption of the Symposium on mind Edema has as its history a compelling necessity to clarify this topic that's clinically so vital and neuropathologic best friend so poorly understood. It was once was hoping that this is able to be completed through a multidisciplinary strategy regarding a number of facets to be mentioned and correlated. mind edema, being an irregular cerebral situation, belongs essentially to the sector of neuropathology, yet to achieve success in our endeavors we needed to observe neuropathology in its latest and widest phrases. through this we take into account that such neuropathology comprises the research of all irregular phenomena within the apprehensive tissue relating to the structural adjustments, and by way of "structural" we suggest not just tissual or mobile ones but in addition adjustments on ultrastructural and molecular degrees. therefore we would have liked shut and built-in coopera tion of specialists in fields resembling neurohistology, histochemistry, electron microscopy, neurochemistry, and neurophysiology.
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Additional info for Brain Edema: Proceedings of the Symposium September 11–13, 1965, Vienna
With successive increments of fluid, however, the baseline of pressure is gradually elevated, and when the intracranial pressure approximates 1200 mm. of water, the blood pressure begins to rise in order to ensure a sufficiency of circulation. If such an inflation is carried out slowly and over a period of only ten to fifteen minutes, deflation of the balloon may result in the return of the pressure to wholly normal limits. If, however, balloon inflation is maintained for a longer period of time and emptying of the balloon is delayed for thirty minutes, there occurs a fall, but shortly thereafter, over the next twenty to thirty minutes, one may observe a steadily rising baseline of pressure (Fig.
F ............ ' ....... \ ~w. nm'lnf. Ii ino. ~ .. , a" THIOPENT (250) IN' Ie' 'at teeners .. "Mnu " ' " , '0 cO'en . ·W Fig. 8. Records demonstrating the effect upon blood pressure and intracranial pressure of barbiturate administration and controlled positive-negative respiration, followed by urea. After injection of a barbiturate (arrow), intracranial pressure (ICP) and blood pressure (BP) fall, and a further fall in ICP is to be noted independent of blood pressure alteration. The amplitude of BP is gradually reduced during controlled respiration (Section 1).
Under these circumstances, it is probable that the swelling was greater in the white than in the grey matter. Of especial clinical interest is the slow time course of partial recovery, even though the hematoma was removed within an hour of injury. Therapeutic efforts consist of supportive measures, including the maintenance of an adequate airway and gas exchange, attention to fluid and electrolyte balance, nutrition, and, where indicated, skilled surgical repair. steroids and hypothermia are thought to be of value in retardation of developing edema, and urea and mannitol are often effective in the reduction of brain volume.