By Helena J. Heuvelman M.D., Nalini Marie Rajamannan M.D. (auth.), Nalini M. Rajamannan (eds.)

The booklet should be written via the specialists within the disorder together with imaging, interventional cardiology and surgical valve services. The ebook has 5 major sections: Aortic Valve ailment Mitral Valve illness Tricuspid Valve sickness Pulmonic Valve affliction Multimodality Imaging the most goals of the e-book are: 1- to supply the most instructions to clinicians on the way to establish and diagnose valvular center sickness 2- to supply an summary of what the close to destiny will herald the analysis, prevention and danger stratification of sufferers to catheter as opposed to surgical methods three- to stipulate the potential implications for clinical cures in slowing development of calcific aortic valve ailment and myxomatous mitral valve disease

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Given its thin walled structure, left atrial size is, however, expected to increase already in earlier stages of AS and be influenced by arterial stiffness, relating to older age and/or concomitant vascular disease such as hypertension. This might suggest that left atrial abnormalities are not good markers of AS severity per se. 37 Notwithstanding, the additional presence of left atrial abnormalities in later AS seems associated with LV and atrial pressure overload (Dalsgaard et al. 2010; Bang et al.

2003b) 4 Experimental Evidence for the Role of Atherosclerosis in Calcific Aortic Valve Disease compared to the non-calcified controls indicating that a regulated bone formation process is occurring at the level of RNA expression. These ex vivo and in vivo models have provided a basic novel understanding of the development of calcification in the aortic valve and may represent parallel signaling pathways that are present in aortic valve myofibroblast cells and osteoblast cells. Furthermore, these studies demonstrate that the aortic valve has an actual bone biology instead of a passive degenerative process.

Heart. 2005a;91(6):806–10. Rajamannan NM, Subramaniam M, Caira F, Stock SR, Spelsberg TC. Atorvastatin inhibits hypercholesterolemia-induced calcification in the aortic valves via the Lrp5 receptor pathway. Circulation. 2005b;112(9 Suppl):I229–34. Schoppet M, Preissner KT, Hofbauer LC. RANK ligand and osteoprotegerin: paracrine regulators of bone metabolism and vascular function. Arterioscler Thromb Vasc Biol. 2002;22(4):549–53. Shao JS, Cheng SL, Pingsterhaus JM, CharltonKachigian N, Loewy AP, Towler DA.

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