By Helen Griffiths

Coeliac affliction: Nursing Care and administration offers nurses with the data and facts base to appreciate the effect of the prognosis of Coeliac sickness, and look at the long run remedy and administration of the . The authors take the reader via an investigative trip from the heritage of the illness via its pathology, features, analysis, therapy and administration. scientific case reviews convey to existence either the actual and psychosocial facets of care of sufferers making this a great textual content for medical nurse experts, Gastroenterology nurses and Endoscopy nurses.

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Additional info for Coeliac Disease: Nursing Care and Management (Wiley Series in Nursing)

Sample text

2001) Current approaches to diagnosis and treatment of celiac disease: An evolving spectrum. Gastroenterology 120:636–651. J. (2006) Hepatobiliary and pancreatic disorders in celiac disease (Editorial). World Journal of Gastroenterology 12(10):1503–1508. M. S. (2005) Atypical presentation of coeliac disease. British Medical Journal 330:773–774. M. A. (1994) 25 years’ experience of a gluten free diet in the treatment of dermatitis herpetiformis. British Journal of Dermatology 131:541–545. Gee, S.

In Pocket Consultant Gastroenterology. Blackwell Publishing Ltd, Oxford, pp. 207–242. Chapter 3 Presentation of Coeliac Disease LEARNING OUTCOMES At the end of this chapter you should be able to: Discuss the changes in disease presentation over time. Describe the more unusual disease presentations. Discuss the implications of screening an unsuspecting population for coeliac disease. Describe the nursing support this group of patients require at this time and how this is best delivered. For many years, coeliac disease has been associated with weight loss, diarrhoea and malabsorption of nutrients.

4 Pathogenesis of coeliac disease. ) 28 Coeliac Disease The presence of a prolamine peptide within the lumen of the small bowel will also cause activation of the innate immune system by the release of a cytokine, interleukin 15 (IL15). Studies suggest that this is in fact a different prolamine peptide than that which induces the activation of the adaptive immune response (involving APCs, HLA-DQ2 and T cells). Because of the enhanced expression of IL15, an MHC-type molecule named MICA is expressed on epithelial enterocytes together with the upregulation of a particular receptor (NKG2D) on intraepithelial lymphocytes (IELs).

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