By Noriyuki Hachiya PhD (auth.), Sandra Ceccatelli, Michael Aschner (eds.)

Mercury (Hg) is an international pollutant that is familiar with no environmental barriers. Even the main stringent keep watch over of anthropogenic Hg resources won't get rid of publicity given its ubiquitous presence. publicity to Hg happens essentially through the meals chain as a result of MeHg’s accumulation in fish. most up-to-date US data point out that forty six States have fish intake advisories. additionally, Hg is a standard pollutant in dangerous waste websites, with an predicted 3-4 million childrens dwelling inside one mile of at the very least one of many 1,300+ energetic damaging waste websites within the US. the consequences on highbrow functionality in childrens prenatally uncovered to MeHg through maternal fish intake were the topic of 2 on-going significant, potential, longitudinal stories within the Seychelles and the Faroe Islands. you will need to realize that the danger for MeHg publicity isn't restricted merely to islanders with excessive fish intake.

This ebook will supply state of the art info to the graduate scholar education in toxicology, threat assessors, researchers and clinical services at huge. it really is aimed to carry the reader modern on modern concerns linked to publicity to methylmercury, from its results on stem cells and neurons to inhabitants reports.

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Extra info for Methylmercury and Neurotoxicity

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Soon after the first use of di-methylmercury in 1860s, two laboratory technicians in London suffered from poisoning and serious central nervous system signs, such as paresthesias of the hands, deafness, poor vision, ataxia, and dysarthria (Hunter et al. 1940). , by poisoning cases in a factory where fungicidal dusts were manufactured. Severe generalized ataxia, dysarthria, and gross constriction of the visual fields were present in the severe cases, with memory and intelligence apparently being unaffected (Hunter et al.

1995b;16:639–52. Myers GJ, Marsh DO, Cox C, et al. A pilot neurodevelopmental study of Seychellois children following in utero exposure to methylmercury from a maternal fish diet. Neurotoxicology. 1995c;16:629–38. Myers GJ, Davidson PW, Shamlaye CF, et al. Effects of prenatal methylmercury exposure from a high fish diet on developmental milestones in the Seychelles child development study. Neurotoxicology. 1997;18:819–29. Myers GJ, Davidson PW, Cox C, et al. Prenatal methylmercury exposure from ocean fish consumption in the Seychelles child development study.

5 mg/g) were compared with 22 controls in a cross-sectional design; the blood concentration of organic mercury was associated with decreased neurological performance in regard to reaction time and motor coordination (Carta et al. 2003). Thus, the possible effects due to low-level exposure to methylmercury in the general population are of considerable concern. 1 mg/g (range: 0–16 mg/g) did not show any clear mercury-related deficits on 20 scores from 12 neurobehavioral tests (Weil et al. 2005). Further studies are required to elucidate the dose–response relationship at low exposure levels.

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